HSPA8 Activates Wnt/β‐Catenin Signaling to Facilitate BRAF V600E Colorectal Cancer Progression by CMA‐Mediated CAV1 Degradation - Li - Advanced Science - Wiley Online Library
HSPA8 Activates Wnt/β‐Catenin Signaling to Facilitate BRAF V600E Colorectal Cancer Progression by CMA‐Mediated CAV1 Degradation - Li - Advanced Science - Wiley Online Library
Signaling pathways in cancer-associated fibroblasts and targeted
Kras promotes myeloid differentiation through Wnt/β‐catenin
Wnt/β-catenin signaling and p68 conjointly regulate CHIP in
Adaptive feedback signaling in BRAF V600E-mutated mCRC. (A) In
Oncogenic BRAF, unrestrained by TGFβ-receptor signalling, drives
Wnt/β-catenin signaling in colorectal cancer: Is therapeutic
Contemporary treatment approaches for metastatic colorectal cancer
Seeking therapeutic synergy in BRAF mutant colorectal cancer
Cancers, Free Full-Text
Wnt/β-catenin signaling and p68 conjointly regulate CHIP in
Wnt/β-catenin signaling and p68 conjointly regulate CHIP in
BRAF-Mutated Advanced Colorectal Cancer: A Rapidly Changing
HSPA8 Activates Wnt/β‐Catenin Signaling to Facilitate BRAF V600E